Pesticides Health Dictionary

Pesticides: From 2 Different Sources


Poisonous chemicals used to eradicate pests. Different types include herbicides, insecticides, and fungicides. Pesticide poisoning, particularly in children, may result from swallowing an insecticide or a garden herbicide (see chlorate poisoning). Poisoning may also occur in agricultural workers, often as a result of inhalation or absorption of the chemical through the skin. Exposure to pesticides can also occur indirectly, through eating food in which chemicals have accumulated as a result of crop spraying. (See also DDT; defoliant poisoning; lindane; paraquat; parathion.)
Health Source: BMA Medical Dictionary
Author: The British Medical Association
Any substance or mixture of substances intended for preventing or controlling unwanted species of plants and animals. This includes any substances intended for use as plant-growth regulators, defoliants or desiccants. The main groups of pesticides are: herbicides to control weeds; insecticides to control insects; and fungicides to control or prevent fungal disease.
Health Source: Medical Dictionary
Author: Health Dictionary

Corn Silk Tea Remedy

Have you ever thought that if you remove the corn silk from corn combs, you can use it as a remedy? While many people may not be familiar with this type of tea, in fact corn silk tea was used for a long time even by Native Americans as a remedy for heart problems, malaria or urinary tract infections. More about Corn silk tea Corn silk is in fact the thin, hair-like strands that cover the corn cob. These silky yellowish strands which form the stigma collect pollen to fertilize the corn, and they’re also used to make a healing tea. In corn silk there can be found many important components like flavonoids, allantoin, mucilage, saponins, vitamins C and K and potassium. Corn silk may also be combined with other herbs to increase its healing powers and range of medicinal uses. It’s also available in prepackaged teabags, or in a dried supplement form. Powdered corn silk is a common ingredient in face powders, due to its soothing qualities. Corn silk tea has a slightly sweet taste. If you decide to collect it in order to make a tea, make sure that the plants were not sprayed with pesticides. Brew corn silk tea In order to make a tasty healthy corn silk tea it is usually recommended to use fresh corn silk. If you don’t have it at your hand, the dried one works just fine. To prepare the infusion, use 2 teaspoons of fresh corn silk or 2.5 g of dried one and pour 1 cup of boiled water over it. Let it seep for 10 - 15 minutes and it is ready to serve. Corn silk tea benefits Corn silk tea has many health benefits for adults and for children. The most important benefit of this tea is for disorders in the urinary system : infections, cystitis, as well as bladder infections or gonorrhea. If you want your children to stop wetting their beds give them corn silk tea. Corn silk tea is also diuretic, demulcent, has anti-inflammatory properties and it fights kidney stones. Corn silk tea may help detoxify and flush out accumulated toxins in the body. Corn silk tea contains vitamin K, which has been shown to improve the body’s blood clotting process. Corn silk tea has also been shown to lower blood pressure, relieve arthritis pains, and help in the treatment of jaundice and prostate disorders. When applied topically, corn silk tea can help heal wounds and skin ulcers. Corn silk tea side effects In most cases, corn silk tea is suitable for daily consumption without special warnings. However, in rare cases, in you are allergic to corn, you may develop a skin rash. Corn silk tea can also decrease the level of potassium in your blood. So you should avoid it if you already have low potassium levels, problems with blood pressure, or diabetes. It is not recommended for children, during pregnancy or breastfeeding. Corn silk tea is safe to be included in your diet, but in order to enjoy its benefits, do not exceed 3 cups a day.... corn silk tea remedy

Gulf War Syndrome

A collection of varying symptoms, such as persistent tiredness, headaches, muscle pain and poor concentration, reported by members of the Coalition Armed Forces who served in the 1991 Gulf War. Whilst there is strong evidence for a health e?ect related to service, there is no evidence of a particular set of signs and symptoms (the de?nition of a ‘syndrome’) unique to those who served in the Gulf War. Symptoms have been blamed on multiple possible hazards, such as exposure to depleted uranium munitions, smoke from oil-well ?res and use of pesticides. However, the only clearly demonstrated association is with the particular pattern of vaccinations used to protect against biological weapons. Many con?icts in the past have generated their own ‘syndromes’, given names such as e?ort syndrome and shell-shock, suggesting a link to the psychological stress of being in the midst of warfare.... gulf war syndrome

Adolescence

This is the time of life when profound physical and emotional changes take place in young people, marking the beginning of puberty and proceeding throughout teenage years towards maturity. It is a time when sound nutrition should bypass many of the distressing crises which arise from heredity tendencies or an unhealthy life-style. Problems of puberty:–

Treatment. Girls. Delayed menarche (Raspberry leaf tea), and other menstrual disorders; hormone deficiency (laboratory tests confirm). Puberty goitre (Kelp), skin disorders: see “Acne”. Listlessness, (Gentian). Loss of appetite (Chamomile). Over-activity, tearfulness, (Pulsatilla).

Boys. Constitutional weaknesses from childhood, (Sarsaparilla); puberty goitre (Kelp); Offensive foot sweat, see: DIURETICS. Aggression, over-activity, (Alfalfa). Under-developed testes (Liquorice, Sarsaparilla).

Nervousness and restlessness of many of the younger generation may arise from a number of causes, including a diet of too much sugar, coffee, caffeine stimulants (coffee, cola, strong tea) and foods deficient in nutrients and minerals. The condition can be related to the number of chemicals used in food and commercial products, pesticides and drugs.

Diet: Plenty fresh raw fruits and vegetables. Raw food days. High protein, low salt, low fat. Alfalfa tea (rich in builder minerals).

Reject: coffee, cola drinks, strong tea, alcohol, tobacco. ... adolescence

Arsenic

A poisonous metallic element that occurs naturally in its pure form and in various compounds. Arsenic poisoning, which is now rare, used to occur as a result of continuous exposure to industrial pesticides.... arsenic

Ddt

The abbreviation for the insecticide dichlorodiphenyltrichloroethane. was once widely used in the fight against diseases that are transmitted by insects, particularly in hot climates. However, some insects have developed resistance, which can be passed on to offspring.

(See also pesticides.)... ddt

Pesticide

n. a chemical agent used to kill insects or other organisms harmful to crops and other cultivated plants. Some pesticides, such as *parathion and *dieldrin, have caused poisoning in human beings and livestock after accidental exposure.... pesticide

Enjoy A Cup Of Rose Petal Tea

It you want to drink a special type of herbal tea, try the rose petal tea. It is aromatic, with a pleasant taste, and you’re bound to enjoy it. It also has important health benefits. Find out more about rose petal tea! About Rose Petal Tea Rose petal tea is made from the petals of a flower most adored by many women: the rose. This woody perennial plant has over 100 species which grow in Asia, Europe, North America, and northwest Africa. Roses grow as a group of erect shrubs, acting like climbing plants. Its stems often have small, sharp thorns. The leaves are oval-shaped with sharply-toothed edges, and they’re about 10cm long. The fruit is called rosehip; it is ripe from late summer to autumn, and it is edible. The flowers usually have 5 petals with two distinct lobes; they are usually pink, white, red, or yellow. You can make tea both from the rose petals and from the rose’s fruit, the rosehip. How to prepare Rose Petal Tea When making rose petal tea, first make sure that the petals you use are free of pesticides. Roses from gardens and flower shops are usually treated with pesticides, and shouldn’t be used to make rose petal tea. To enjoy rose petal tea, add about two handfuls of properly washed and dry rose petals to a pot with water for three cups of tea. Leave the pot over medium heat for 5 minutes, or until the petals have lost their original color, becoming darker. Stream to remove the petals and sweeten, if necessary, with honey or fruit juice. Rose Petal Tea Components Rose petal tea gets many active components from the rose petals: cyclic monoterpene alcohols, geraniol, citronellol and nerol are just a few important ones. It also includes long-chain hydrocarbons (nonadecane, heneicosane). These active components lead to the many health benefits rose petal tea has. Rose Petal Tea Benefits Rose petal tea helps strengthen your immunity, and can be part of the treatment for colds. It is useful if you’ve got a fever, a runny nose, a sore throat, or bronchial congestion. Also, it helps clean your body of toxins. Drinking rose petal tea can help during menstrual periods, if you’ve got a heavy menstrual flow. It can also reduce menstrual cramps, and helps regulate your period. Rose petal tea is often used to treat diarrhea and dysentery. It can also help you fight against depression, fatigue and insomnia. Rose petal tea also acts as a digestive aid, as it protects the gastrointestinal tract. It is often used to treat constipation, gastroenteritis, diarrhea, and dysentery; the tea also nourishes the gastric mucosa. You can drink rose petal tea to treat urinary tract infections, as well. Rose Petal Tea Side Effects No important side effects of rose petal tea have been noted. Still, it is considered best not to drink more than 5 cups of tea a day. If you drink too much, you might get some of these symptoms: headaches, loss of appetite, vomiting, diarrhea, insomnia, dizziness, and irregular heartbeats. Spoil yourself with a delicious cup of rose petal tea! Not only will you enjoy its taste, but its health benefits, as well.... enjoy a cup of rose petal tea

Herbs, Source Of

Today’s practice: only first grade organically grown herbs (European, British, American, etc) without the aid of pesticides or herbicides are the general rule, but standards vary in different countries. Some herbs are freshly picked and processed on the same day, and it is modern practice to cultivate in a remote location to avoid wind-blown chemical contamination.

Herbs are soft-stemmed plants that die back in winter. No artificial additives; no cruelty to animals; and no damage to the environment is the ideal in the preparation of herbal remedies.

All herbs are subject to natural variations such as weather, climate and constituents of the soil. In herbal pharmacy products are standardised as carefully as possible under strict laboratory conditions. ... herbs, source of

Insects And Disease

Relatively few insect species cause disease directly in humans. Some parasitize humans, living under the skin or on the body surface (see lice; chigoe; myiasis). The most troublesome insects are flies and biting insects. Flies can carry disease organisms from human or animal excrement via their feet or legs and contaminate food or wounds.

A number of serious diseases are spread by biting insects.

These include malaria and filariasis (transmitted by mosquitoes), sleeping sickness (tsetse flies), leishmaniasis (sandflies), epidemic typhus (lice), and plague (rat fleas).

Mosquitoes, sandflies, and ticks can also spread illnesses such as yellow fever, dengue, Lyme disease, and some types of viral encephalitis.

Organisms picked up when an insect ingests blood from an infected animal or person are able to survive or multiply in the insect.

Later, the organisms are either injected into a new human host via the insect’s saliva or deposited in the faeces at or near the site of the bite.

Most insect-borne diseases are confined to the tropics and subtropics, although tick-borne Lyme disease occurs in some parts of the.

The avoidance of insect-borne disease is largely a matter of keeping flies off food, discouraging insect bites by the use of suitable clothing and insect repellents, and, in parts of the world where malaria is present, the use of mosquito nets and screens, pesticides, and antimalarial tablets.... insects and disease

Environment And Health

Environment and Health concerns those aspects of human health, including quality of life, that are determined by physical, biological, social and psychosocial factors in the environment. The promotion of good health requires not only public policies which support health, but also the creation of supportive environments in which living and working conditions are safe, stimulating and enjoyable.

Health has driven much of environmental policy since the work of Edwin Chadwick in the early 1840s. The ?rst British public-health act was introduced in 1848 to improve housing and sanitation with subsequent provision of puri?ed water, clean milk, food hygiene regulations, vaccinations and antibiotics. In the 21st century there are now many additional environmental factors that must be monitored, researched and controlled if risks to human health are to be well managed and the impact on human morbidity and mortality reduced.

Environmental impacts on health include:

noise

air pollution

water pollution

dust •odours

contaminated ground

loss of amenities

vermin

vibration

animal diseases

Environmental risk factors Many of the major determinants of health, disease and death are environmental risk factors. Some are natural hazards; others are generated by human activities. They may be directly harmful, as in the examples of exposure to toxic chemicals at work, pesticides, or air pollution from road transport, or to radon gas penetrating domestic properties. Environmental factors may also alter people’s susceptibility to disease: for example, the availability of su?cient food. In addition, they may operate by making unhealthy choices more likely, such as the availability and a?ord-ability of junk foods, alcohol, illegal drugs or tobacco.

Populations at risk Children are among the populations most sensitive to environmental health hazards. Their routine exposure to toxic chemicals in homes and communities can put their health at risk. Central to the ability to protect communities and families is the right of people to know about toxic substances. For many, the only source of environmental information is media reporting, which often leaves the public confused and frustrated. To bene?t from public access to information, increasingly via the Internet, people need basic environmental and health information, resources for interpreting, understanding and evaluating health risks, and familiarity with strategies for prevention or reduction of risk.

Risk assessment Environmental health experts rely on the principles of environmental toxicology and risk assessment to evaluate the environment and the potential effects on individual and community health. Key actions include:

identifying sources and routes of environmental exposure and recommending methods of reducing environmental health risks, such as exposure to heavy metals, solvents, pesticides, dioxins, etc.

assessing the risks of exposure-related health hazards.

alerting health professionals, the public, and the media to the levels of risk for particular potential hazards and the reasons for interventions.

ensuring that doctors and scientists explain the results of environmental monitoring studies – for example, the results of water ?uoridation in the UK to improve dental health.

National policies In the United Kingdom in 1996, an important step in linking environment and health was taken by a government-initiated joint consultation by the Departments of Health and Environment about adding ‘environment’ as a key area within the Health of the Nation strategy. The ?rst UK Minister of State for Public Health was appointed in 1997 with responsibilities for health promotion and public-health issues, both generally and within the NHS. These responsibilities include the implementation of the Health of the Nation strategy and its successor, Our Healthy Nation. The aim is to raise the priority given to human health throughout government departments, and to make health and environmental impact assessment a routine part of the making, implementing and assessing the impact of policies.

Global environmental risks The scope of many environmental threats to human health are international and cannot be regulated e?ectively on a local, regional or even national basis. One example is the Chernobyl nuclear reactor accident, which led to a major release of radiation, the effects of which were felt in many countries. Some international action has already been taken to tackle global environmental problems, but governments should routinely measure the overall impacts of development on people and their environments and link with industry to reduce damage to the environment. For instance, the effects of global warming and pollution on health should be assessed within an ecological framework if communities are to respond e?ectively to potential new global threats to the environment.... environment and health

Neem

Azadirachta indica

Meliaceae

San: Nimbah, Prabhadrah Hin,

Ben: Nim, Nim Mal: Aryaveppu

Tel: Vepa Ori: Nimba

Tam: Vembu, Veppu Pun: Bakam,Bukhain

Guj: Limba

Kan: Bevu Mar: Limbu

Importance: Neem or margose tree, also known as Indian lilac is a highly exploited medicinal plant of Indian origin, widely grown and cultivated throughout India. Every part of the tree, namely root, bark, wood, twig, leaf, flower, fruit, seed, kernel and oil has been in use from time immemorial in the Ayurvedic and Unani systems of medicine. Nimbarishta, nimbadi churna and nimbharidra khand are well known preparations. It is valuable as an antiseptic, used in the treatment of small pox. Small twigs are used as tooth brushes and as a prophylactic for mouth and teeth complaints. Extract from the leaves are useful for sores, eczema and skin diseases. Boiled and smashed leaves serve as excellent antiseptic. Decoction of leaves is used for purifying blood. Neem oil is used in soaps, toothpaste and as a hair tonic to kill lice. Seed is used in snake bite. The fruits and leaves being renewable, provide sustainable returns. Different parts of the fruit are separated into components and each one produces derivatives of varying chemical nature and utility. Neem derivatives are now used in agriculture, public health, human and veterinary medicines, toiletries, cosmetics and livestock production. Applications as pesticides, allied agrochemicals, plant nutrients and adjuvants for improving nitrogen use efficiency are of much importance. Neem kernel suspension (1%) is a house hold insecticide. Pesticide formulations containing azadirachtin are now commercially available in India, USA, Canada, Australia and Germany. Neem cake is rich in N, P, K, Ca and S. Neem Meliacins like epinimbin and nimbidin are commercially exploited for the preparation of slow and extended release of nutrients including nitrification inhibitors (Eg. Nimin). Extracts of neem seed oil and bark check the activity of male reproductive cells and prevents sperm production. Neem seed oil is more effective than the bark for birth control. Neem based commercial products are also available for diabetes treatment (Nimbola, JK-22), contraceptive effect (Sensal, Nim-76) and mosquito/ insect repelling (Srivastava, 1989; Tewari, 1992; Parmer and Katkar, 1993; Pushpangadan et al, 1993; Mariappan, 1995).

Distribution: Neem is a native of the Siwalik deccan parts of South India. It grows wild in the dry forests of Andra Pradesh, Tamil Nadu and Karnataka. It has spread to Pakistan, Bangladesh , Sri Lanka, Malaysia, Indonesia, Thailand, Middle East Sudan and Niger. It is now grown in Australia, Africa, Fiji, Mauritious, Central and South America, the Carribeans, Puerto Rico and Haiti. The largest known plantation of nearly 50,000 trees is at Arafat plains en route to Mecca in Saudi Arabia for providing shade to Haj pilgrims (Ahmed, 1988).

Botany: The genus Azadirachta of family Meliaceae comprises two species: A. indica A. Juss syn. Melia azadirachta Linn. and A. excelsa (Jack) Jacobs syn. A. integrifolia Mers., the latter being found in Philippines, Sumatra, Malaya, Borneo and New Guinea. Neem is a hardy medium to large, mostly evergreen tree attaining 20m height and 2.5m girth. It has a short bole with wide spreading branches and glabrous twigs forming a round to oval crown. The bark is thick, dark-gray with numerous longitudinal furrows and transverse cracks. Leaves are imparipinnately compound, alternate, exstipulate and 20-38cm long. Inflorescence is long, slender, axillary or terminal panicle. Flowers are white or pale yellow, small, bisexual, pentamerous and bracteate. Stamens 10; filaments unite to form a moniliform tube. Gynoecium is tricarpellary and syncarpous, ovary superior, trilocular. Each carpel bears two collateral ovules on parietal placentation. Fruit is one seeded drupe with woody endocarp, greenish yellow when ripe. Seed ellipsoid, cotyledons thick fleshy and oily. Neem has chromosome number 2n = 28. Neem trees tend to become deciduous for a brief period in dry ecology. Ecotypes, exhibiting morphological variation in root growth, leaf size, contents, bole length , canopy, inflorescence, fruit bearing, seed size, shape and quality exist in natural populations.

Agrotechnology: Neem grows in tropical arid regions with high temperatures, altitudes between 50m and 1000m, as little rainfall as 130mm/yr and long stretches of drought. Well drained sunny hill places are ideal. It grows on most kinds of soils including dry, stony, shallow, nutrient deficient soils with scanty vegetation, moderately saline and alkali soils, black cotton, compact clays and laterite crusts. However, silty flats, clayey depressions and land prone to inundation are not conducive for its growth (Chaturvedi, 1993). Soil pH of 5.0 to 10.0 is ideal. It brings surface soil to neutral pH by its leaf litter. It has extensive and deeply penetrating root system capable of extracting moisture and nutrients even from highly leached poor sandy soils.

Neem propagates easily by seed without any pretreatment, though it can be regenerated by vegetative means like root and shoot cuttings. Seeds are collected from June to August. These remain viable for 3-5 weeks only which necessitates sowing within this short time. Seeds may be depulped and soaked in water for 6 hours before sowing. Seeds are sown on nursery beds at 15x5cm spacing, covered with rotten straw and irrigated. Germination takes 15-30 days. Seedlings can be transplanted after two months of growth onwards either to polybags or to mainfield. Neem can be grown along with agricultural crops like groundnut, bean, millets, sorghum and wheat. It is also suitable for planting in roadsides, for afforestation of wastelands and under agroforestry system. For field planting, pits of size 50-75 cm cube are dug 5-6m apart, filled with top soil and well rotten manure, formed into a heap, and seedling is planted at the centre of the heap. FYM is applied at 10-20 kg/plant every year. Chemical fertilizers are not generally applied. Irrigation and weeding are required during the first year for quick establishment.

More than 38 insect pests are reported on Neem which may become serious at times. The important ones are seed and flower insect (Scirtothrips dorsalis Hood), defoliators (Boarmia variegata Moore and Eurema sp.), sap suckers (Helopeltes antonii Signoret and Pulvinaria maxima Green) , root feeders (Hototrichia consanguinea Blanchard), mealy bug (Pseudococus gilbertensis), scale insect (Parlatoria orientalis) and a leaf webber (Loboschiza Koenigiana)(Beeson, 1941, Bhasin et al, 1958, Parmar, 1995). They can be controlled by the application of 0.01-0.02% monocrotophos or dimethoate. No serious diseases are reported in Neem. Flowering starts after 5 years. In India flowering is during January-May and fruits mature from May-August. The leaves are shed during February- March and a full grown tree produces about 350 kg dry leaves and 40-50 kg berries per annum. Fresh fruits give 60% dry fruits which yield 10% kernel which contains 45% fixed oil, on an average. After 10 years of growth the wood can be cut and used as timber.

Properties and Activity: Dry Neem leaves contain carbohydrates 47-51%, crude protein 14-19%, crude fiber 11-24%, fat 2-7%, ash 7-9%, Ca 0.8-2.5% and P 0.1-0.2%. Leaves also contain the flavanoid quercetin, nimbosterol (-sitosterol), kaempferol and myricetin. Seed and oil contains desacetylnimbin, azadirachtin (C35H44O16), nimbidol, meliantriol ,tannic acid, S and amino acids. Neem cake contain the highest sulphur content of 1.07% among all the oil cakes. Trunk bark contains nimbin 0.04%, nimbinin 0.001%, nimbidin 0.4%, nimbosterol 0.03%, essential oil 0.02%, tannins 6.0 %, margosine and desacetylnimbin (Atal and Kapur, 1982; Thakur et al 1989).

Neem bark is bitter, astringent, acrid, refrigerant, depurative, antiperiodic, vulnerary, demulcent, insecticidal, liver tonic, expectorant and anthelmintic. Leaves are bitter, astringent, acrid, depurative, antiseptic, ophthalmic, anthelmintic, alexeteric, appetizer, insecticidal, demulcent and refrigerant. Seed and oil are bitter, acrid, thermogenic, purgative, emollient, anodyne, anthelmintic depurative, vulnerary, uterine stimulant, urinary astringent, pesticidal and antimicrobial (Warrier et al, 1993).... neem

Occupational Health, Medicine And Diseases

Occupational health The e?ect of work on human health, and the impact of workers’ health on their work. Although the term encompasses the identi?cation and treatment of speci?c occupational diseases, occupational health is also an applied and multidisciplinary subject concerned with the prevention of occupational ill-health caused by chemical, biological, physical and psychosocial factors, and the promotion of a healthy and productive workforce.

Occupational health includes both mental and physical health. It is about compliance with health-and-safety-at-work legislation (and common law duties) and about best practice in providing work environments that reduce risks to health and safety to lowest practicable levels. It includes workers’ ?tness to work, as well as the management of the work environment to accommodate people with disabilities, and procedures to facilitate the return to work of those absent with long-term illness. Occupational health incorporates several professional groups, including occupational physicians, occupational health nurses, occupational hygienists, ergonomists, disability managers, workplace counsellors, health-and-safety practitioners, and workplace physiotherapists.

In the UK, two key statutes provide a framework for occupational health: the Health and Safety at Work, etc. Act 1974 (HSW Act); and the Disability Discrimination Act 1995 (DDA). The HSW Act states that employers have a duty to protect the health, safety and welfare of their employees and to conduct their business in a way that does not expose others to risks to their health and safety. Employees and self-employed people also have duties under the Act. Modern health-and-safety legislation focuses on assessing and controlling risk rather than prescribing speci?c actions in di?erent industrial settings. Various regulations made under the HSW Act, such as the Control of Substances Hazardous to Health Regulations, the Manual Handling Operations Regulations and the Noise at Work Regulations, set out duties with regard to di?erent risks, but apply to all employers and follow the general principles of risk assessment and control. Risks should be controlled principally by removing or reducing the hazard at source (for example, by substituting chemicals with safer alternatives, replacing noisy machinery, or automating tasks to avoid heavy lifting). Personal protective equipment, such as gloves and ear defenders, should be seen as a last line of defence after other control measures have been put in place.

The employment provisions of the DDA require employers to avoid discriminatory practice towards disabled people and to make reasonable adjustments to working arrangements where a disabled person is placed at a substantial disadvantage to a non-disabled person. Although the DDA does not require employers to provide access to rehabilitation services – even for those injured or made ill at work – occupational-health practitioners may become involved in programmes to help people get back to work after injury or long-term illness, and many businesses see the retention of valuable sta? as an attractive alternative to medical retirement or dismissal on health grounds.

Although a major part of occupational-health practice is concerned with statutory compliance, the workplace is also an important venue for health promotion. Many working people rarely see their general practitioner and, even when they do, there is little time to discuss wider health issues. Occupational-health advisers can ?ll in this gap by providing, for example, workplace initiatives on stopping smoking, cardiovascular health, diet and self-examination for breast and testicular cancers. Such initiatives are encouraged because of the perceived bene?ts to sta?, to the employing organisation and to the wider public-health agenda. Occupational psychologists recognise the need for the working population to achieve a ‘work-life balance’ and the promotion of this is an increasing part of occupational health strategies.

The law requires employers to consult with their sta? on health-and-safety matters. However, there is also a growing understanding that successful occupational-health management involves workers directly in the identi?cation of risks and in developing solutions in the workplace. Trade unions play an active role in promoting occupational health through local and national campaigns and by training and advising elected workplace safety representatives.

Occupational medicine The branch of medicine that deals with the control, prevention, diagnosis, treatment and management of ill-health and injuries caused or made worse by work, and with ensuring that workers are ?t for the work they do.

Occupational medicine includes: statutory surveillance of workers’ exposure to hazardous agents; advice to employers and employees on eliminating or reducing risks to health and safety at work; diagnosis and treatment/management of occupational illness; advice on adapting the working environment to suit the worker, particularly those with disabilities or long-term health problems; and advice on the return to work and, if necessary, rehabilitation of workers absent through illness. Occupational physicians may play a wider role in monitoring the health of workplace populations and in advising employers on controlling health hazards where ill-health trends are observed. They may also conduct epidemiological research (see EPIDEMIOLOGY) on workplace diseases.

Because of the occupational physician’s dual role as adviser to both employer and employee, he or she is required to be particularly diligent with regards to the individual worker’s medical CONFIDENTIALITY. Occupational physicians need to recognise in any given situation the context they are working in, and to make sure that all parties are aware of this.

Occupational medicine is a medical discipline and thus is only part of the broader ?eld of occupational health. Although there are some speci?c clinical duties associated with occupational medicine, such as diagnosis of occupational disease and medical screening, occupational physicians are frequently part of a multidisciplinary team that might include, for example, occupational-health nurses, healthand-safety advisers, ergonomists, counsellors and hygienists. Occupational physicians are medical practitioners with a post-registration quali?cation in occupational medicine. They will have completed a period of supervised in-post training. In the UK, the Faculty of Occupational Medicine of the Royal College of Physicians has three categories of membership, depending on quali?cations and experience: associateship (AFOM); membership (MFOM); and fellowship (FFOM).

Occupational diseases Occupational diseases are illnesses that are caused or made worse by work. In their widest sense, they include physical and mental ill-health conditions.

In diagnosing an occupational disease, the clinician will need to examine not just the signs and symptoms of ill-health, but also the occupational history of the patient. This is important not only in discovering the cause, or causes, of the disease (work may be one of a number of factors), but also in making recommendations on how the work should be modi?ed to prevent a recurrence – or, if necessary, in deciding whether or not the worker is able to return to that type of work. The occupational history will help in deciding whether or not other workers are also at risk of developing the condition. It will include information on:

the nature of the work.

how the tasks are performed in practice.

the likelihood of exposure to hazardous agents (physical, chemical, biological and psychosocial).

what control measures are in place and the extent to which these are adhered to.

previous occupational and non-occupational exposures.

whether or not others have reported similar symptoms in relation to the work. Some conditions – certain skin conditions,

for example – may show a close relationship to work, with symptoms appearing directly only after exposure to particular agents or possibly disappearing at weekends or with time away from work. Others, however, may be chronic and can have serious long-term implications for a person’s future health and employment.

Statistical information on the prevalence of occupational disease in the UK comes from a variety of sources, including o?cial ?gures from the Industrial Injuries Scheme (see below) and statutory reporting of occupational disease (also below). Neither of these o?cial schemes provides a representative picture, because the former is restricted to certain prescribed conditions and occupations, and the latter suffers from gross under-reporting. More useful are data from the various schemes that make up the Occupational Diseases Intelligence Network (ODIN) and from the Labour Force Survey (LFS). ODIN data is generated by the systematic reporting of work-related conditions by clinicians and includes several schemes. Under one scheme, more than 80 per cent of all reported diseases by occupational-health physicians fall into just six of the 42 clinical disease categories: upper-limb disorders; anxiety, depression and stress disorders; contact DERMATITIS; lower-back problems; hearing loss (see DEAFNESS); and ASTHMA. Information from the LFS yields a similar pattern in terms of disease frequency. Its most recent survey found that over 2 million people believed that, in the previous 12 months, they had suffered from an illness caused or made worse by work and that

19.5 million working days were lost as a result. The ten most frequently reported disease categories were:

stress and mental ill-health (see MENTAL ILLNESS): 515,000 cases.

back injuries: 508,000.

upper-limb and neck disorders: 375,000.

lower respiratory disease: 202,000.

deafness, TINNITUS or other ear conditions: 170,000.

lower-limb musculoskeletal conditions: 100,000.

skin disease: 66,000.

headache or ‘eyestrain’: 50,000.

traumatic injury (includes wounds and fractures from violent attacks at work): 34,000.

vibration white ?nger (hand-arm vibration syndrome): 36,000. A person who develops a chronic occu

pational disease may be able to sue his or her employer for damages if it can be shown that the employer was negligent in failing to take reasonable care of its employees, or had failed to provide a system of work that would have prevented harmful exposure to a known health hazard. There have been numerous successful claims (either awarded in court, or settled out of court) for damages for back and other musculoskeletal injuries, hand-arm vibration syndrome, noise-induced deafness, asthma, dermatitis, MESOTHELIOMA and ASBESTOSIS. Employers’ liability (workers’ compensation) insurers are predicting that the biggest future rise in damages claims will be for stress-related illness. In a recent study, funded by the Health and Safety Executive, about 20 per cent of all workers – more than 5 million people in the UK – claimed to be ‘very’ or ‘extremely’ stressed at work – a statistic that is likely to have a major impact on the long-term health of the working population.

While victims of occupational disease have the right to sue their employers for damages, many countries also operate a system of no-fault compensation for the victims of prescribed occupational diseases. In the UK, more than 60 diseases are prescribed under the Industrial Injuries Scheme and a person will automatically be entitled to state compensation for disability connected to one of these conditions, provided that he or she works in one of the occupations for which they are prescribed. The following short list gives an indication of the types of diseases and occupations prescribed under the scheme:

CARPAL TUNNEL SYNDROME connected to the use of hand-held vibrating tools.

hearing loss from (amongst others) use of pneumatic percussive tools and chainsaws, working in the vicinity of textile manufacturing or woodworking machines, and work in ships’ engine rooms.

LEPTOSPIROSIS – infection with Leptospira (various listed occupations).

viral HEPATITIS from contact with human blood, blood products or other sources of viral hepatitis.

LEAD POISONING, from any occupation causing exposure to fumes, dust and vapour from lead or lead products.

asthma caused by exposure to, among other listed substances, isocyanates, curing agents, solder ?ux fumes and insects reared for research.

mesothelioma from exposure to asbestos.

In the UK, employers and the self-employed have a duty to report all occupational injuries (if the employee is o? work for three days or more as a result), diseases or dangerous incidents to the relevant enforcing authority (the Health and Safety Executive or local-authority environmental-health department) under the Reporting of Injuries, Diseases and Dangerous Occurrences Regulations 1995 (RIDDOR). Despite this statutory duty, comparatively few diseases are reported so that ?gures generated from RIDDOR reports do not give a useful indication of the scale of occupational diseases in the UK. The statutory reporting of injuries is much better, presumably because of the clear and acute relationship between a workplace accident and the resultant injury. More than 160,000 injuries are reported under RIDDOR every year compared with just 2,500 or so occupational diseases, a gross underestimate of the true ?gure.

There are no precise ?gures for the number of people who die prematurely because of work-related ill-health, and it would be impossible to gauge the exact contribution that work has on, for example, cardiovascular disease and cancers where the causes are multifactorial. The toll would, however, dwarf the number of deaths caused by accidents at work. Around 250 people are killed by accidents at work in the UK each year – mesothelioma, from exposure to asbestos at work, alone kills more than 1,300 people annually.

The following is a sample list of occupational diseases, with brief descriptions of their aetiologies.

Inhaled materials

PNEUMOCONIOSIS covers a group of diseases which cause ?brotic lung disease following the inhalation of dust. Around 250–300 new cases receive bene?t each year – mostly due to coal dust with or without silica contamination. SILICOSIS is the more severe disease. The contraction in the size of the coal-mining industry as well as improved dust suppression in the mines have diminished the importance of this disease, whereas asbestos-related diseases now exceed 1,000 per year. Asbestos ?bres cause a restrictive lung disease but also are responsible for certain malignant conditions such as pleural and peritoneal mesothelioma and lung cancer. The lung-cancer risk is exacerbated by cigarette-smoking.

Even though the use of asbestos is virtually banned in the UK, many workers remain at risk of exposure because of the vast quantities present in buildings (much of which is not listed in building plans). Carpenters, electricians, plumbers, builders and demolition workers are all liable to exposure from work that disturbs existing asbestos. OCCUPATIONAL ASTHMA is of increasing importance – not only because of the recognition of new allergic agents (see ALLERGY), but also in the number of reported cases. The following eight substances are most frequently linked to occupational asthma (key occupations in brackets): isocyanates (spray painters, electrical processors); ?our and grain (bakers and farmers); wood dust (wood workers); glutaraldehyde (nurses, darkroom technicians); solder/colophony (welders, electronic assembly workers); laboratory animals (technicians, scientists); resins and glues (metal and electrical workers, construction, chemical processors); and latex (nurses, auxiliaries, laboratory technicians).

The disease develops after a short, symptomless period of exposure; symptoms are temporally related to work exposures and relieved by absences from work. Removal of the worker from exposure does not necessarily lead to complete cessation of symptoms. For many agents, there is no relationship with a previous history of ATOPY. Occupational asthma accounts for about 10 per cent of all asthma cases. DERMATITIS The risk of dermatitis caused by an allergic or irritant reaction to substances used or handled at work is present in a wide variety of jobs. About three-quarters of cases are irritant contact dermatitis due to such agents as acids, alkalis and solvents. Allergic contact dermatitis is a more speci?c response by susceptible individuals to a range of allergens (see ALLERGEN). The main occupational contact allergens include chromates, nickel, epoxy resins, rubber additives, germicidal agents, dyes, topical anaesthetics and antibiotics as well as certain plants and woods. Latex gloves are a particular cause of occupational dermatitis among health-care and laboratory sta? and have resulted in many workers being forced to leave their profession through ill-health. (See also SKIN, DISEASES OF.)

Musculoskeletal disorders Musculoskeletal injuries are by far the most common conditions related to work (see LFS ?gures, above) and the biggest cause of disability. Although not all work-related, musculoskeletal disorders account for 36.5 per cent of all disabilities among working-age people (compared with less than 4 per cent for sight and hearing impairment). Back pain (all causes – see BACKACHE) has been estimated to cause more than 50 million days lost every year in sickness absence and costs the UK economy up to £5 billion annually as a result of incapacity or disability. Back pain is a particular problem in the health-care sector because of the risk of injury from lifting and moving patients. While the emphasis should be on preventing injuries from occurring, it is now well established that the best way to manage most lower-back injuries is to encourage the patient to continue as normally as possible and to remain at work, or to return as soon as possible even if the patient has some residual back pain. Those who remain o? work on long-term sick leave are far less likely ever to return to work.

Aside from back injuries, there are a whole range of conditions affecting the upper limbs, neck and lower limbs. Some have clear aetiologies and clinical signs, while others are less well de?ned and have multiple causation. Some conditions, such as carpal tunnel syndrome, are prescribed diseases in certain occupations; however, they are not always caused by work (pregnant and older women are more likely to report carpal tunnel syndrome irrespective of work) and clinicians need to be careful when assigning work as the cause without ?rst considering the evidence. Other conditions may be revealed or made worse by work – such as OSTEOARTHRITIS in the hand. Much attention has focused on injuries caused by repeated movement, excessive force, and awkward postures and these include tenosynovitis (in?ammation of a tendon) and epicondylitis. The greatest controversy surrounds upper-limb disorders that do not present obvious tissue or nerve damage but nevertheless give signi?cant pain and discomfort to the individual. These are sometimes referred to as ‘repetitive strain injury’ or ‘di?use RSI’. The diagnosis of such conditions is controversial, making it di?cult for sufferers to pursue claims for compensation through the courts. Psychosocial factors, such as high demands of the job, lack of control and poor social support at work, have been implicated in the development of many upper-limb disorders, and in prevention and management it is important to deal with the psychological as well as the physical risk factors. Occupations known to be at particular risk of work-related upper-limb disorders include poultry processors, packers, electronic assembly workers, data processors, supermarket check-out operators and telephonists. These jobs often contain a number of the relevant exposures of dynamic load, static load, a full or excessive range of movements and awkward postures. (See UPPER LIMB DISORDERS.)

Physical agents A number of physical agents cause occupational ill-health of which the most important is occupational deafness. Workplace noise exposures in excess of 85 decibels for a working day are likely to cause damage to hearing which is initially restricted to the vital frequencies associated with speech – around 3–4 kHz. Protection from such noise is imperative as hearing aids do nothing to ameliorate the neural damage once it has occurred.

Hand-arm vibration syndrome is a disorder of the vascular and/or neural endings in the hands leading to episodic blanching (‘white ?nger’) and numbness which is exacerbated by low temperature. The condition, which is caused by vibrating tools such as chain saws and pneumatic hammers, is akin to RAYNAUD’S DISEASE and can be disabling.

Decompression sickness is caused by a rapid change in ambient pressure and is a disease associated with deep-sea divers, tunnel workers and high-?ying aviators. Apart from the direct effects of pressure change such as ruptured tympanic membrane or sinus pain, the more serious damage is indirectly due to nitrogen bubbles appearing in the blood and blocking small vessels. Central and peripheral nervous-system damage and bone necrosis are the most dangerous sequelae.

Radiation Non-ionising radiation from lasers or microwaves can cause severe localised heating leading to tissue damage of which cataracts (see under EYE, DISORDERS OF) are a particular variety. Ionising radiation from radioactive sources can cause similar acute tissue damage to the eyes as well as cell damage to rapidly dividing cells in the gut and bone marrow. Longer-term effects include genetic damage and various malignant disorders of which LEUKAEMIA and aplastic ANAEMIA are notable. Particular radioactive isotopes may destroy or induce malignant change in target organs, for example, 131I (thyroid), 90Sr (bone). Outdoor workers may also be at risk of sunburn and skin cancers. OTHER OCCUPATIONAL CANCERS Occupation is directly responsible for about 5 per cent of all cancers and contributes to a further 5 per cent. Apart from the cancers caused by asbestos and ionising radiation, a number of other occupational exposures can cause human cancer. The International Agency for Research on Cancer regularly reviews the evidence for carcinogenicity of compounds and industrial processes, and its published list of carcinogens is widely accepted as the current state of knowledge. More than 50 agents and processes are listed as class 1 carcinogens. Important occupational carcinogens include asbestos (mesothelioma, lung cancer); polynuclear aromatic hydrocarbons such as mineral oils, soots, tars (skin and lung cancer); the aromatic amines in dyestu?s (bladder cancer); certain hexavalent chromates, arsenic and nickel re?ning (lung cancer); wood and leather dust (nasal sinus cancer); benzene (leukaemia); and vinyl chloride monomer (angiosarcoma of the liver). It has been estimated that elimination of all known occupational carcinogens, if possible, would lead to an annual saving of 5,000 premature deaths in Britain.

Infections Two broad categories of job carry an occupational risk. These are workers in contact with animals (farmers, veterinary surgeons and slaughtermen) and those in contact with human sources of infection (health-care sta? and sewage workers).

Occupational infections include various zoonoses (pathogens transmissible from animals to humans), such as ANTHRAX, Borrelia burgdorferi (LYME DISEASE), bovine TUBERCULOSIS, BRUCELLOSIS, Chlamydia psittaci, leptospirosis, ORF virus, Q fever, RINGWORM and Streptococcus suis. Human pathogens that may be transmissible at work include tuberculosis, and blood-borne pathogens such as viral hepatitis (B and C) and HIV (see AIDS/HIV). Health-care workers at risk of exposure to infected blood and body ?uids should be immunised against hapatitis B.

Poisoning The incidence of occupational poisonings has diminished with the substitution of noxious chemicals with safer alternatives, and with the advent of improved containment. However, poisonings owing to accidents at work are still reported, sometimes with fatal consequences. Workers involved in the application of pesticides are particularly at risk if safe procedures are not followed or if equipment is faulty. Exposure to organophosphate pesticides, for example, can lead to breathing diffculties, vomiting, diarrhoea and abdominal cramps, and to other neurological effects including confusion and dizziness. Severe poisonings can lead to death. Exposure can be through ingestion, inhalation and dermal (skin) contact.

Stress and mental health Stress is an adverse reaction to excessive pressures or demands and, in occupational-health terms, is di?erent from the motivational impact often associated with challenging work (some refer to this as ‘positive stress’). Stress at work is often linked to increasing demands on workers, although coping can often prevent the development of stress. The causes of occupational stress are multivariate and encompass job characteristics (e.g. long or unsocial working hours, high work demands, imbalance between e?ort and reward, poorly managed organisational change, lack of control over work, poor social support at work, fear of redundancy and bullying), as well as individual factors (such as personality type, personal circumstances, coping strategies, and availability of psychosocial support outside work). Stress may in?uence behaviours such as smoking, alcohol consumption, sleep and diet, which may in turn affect people’s health. Stress may also have direct effects on the immune system (see IMMUNITY) and lead to a decline in health. Stress may also alter the course and response to treatment of conditions such as cardiovascular disease. As well as these general effects of stress, speci?c types of disorder may be observed.

Exposure to extremely traumatic incidents at work – such as dealing with a major accident involving multiple loss of life and serious injury

(e.g. paramedics at the scene of an explosion or rail crash) – may result in a chronic condition known as post-traumatic stress disorder (PTSD). PTSD is an abnormal psychological reaction to a traumatic event and is characterised by extreme psychological discomfort, such as anxiety or panic when reminded of the causative event; sufferers may be plagued with uncontrollable memories and can feel as if they are going through the trauma again. PTSD is a clinically de?ned condition in terms of its symptoms and causes and should not be used to include normal short-term reactions to trauma.... occupational health, medicine and diseases

Eczema

The most common skin disease; recognised by minute blisters (vesicles) which fill with colourless fluid and burst leaving the skin cracked, scaly and weepy with possible bleeding. Successful treatment depends upon recognising the type and distribution. Partly a metabolic imbalance.

Atopic eczema. Allergic eczema. May run in families together with hay fever, asthma or inflamed nasal membrane. May appear anywhere but prefers elbows, knees (flexures), ankles or face. Often seen in infants. May return again and again throughout adult life. Scratching exacerbates.

As regards babies, some paediatricians believe breast-feeding to be protective. A stronger case follows investigation into pollutants from the atmosphere or as additives in food. Industrial chemicals find their way into breast milk that may not be easily excreted but stored in fat.

Cow’s milk is particularly suspect because of exposure of the animal to herbicides and pesticides. For this reason, goat’s milk has met with some success in treatment of this condition, as has Soya milk. Now known that food plays an important part in effective treatment. Chief allergy-stimulators: dairy produce, eggs, cow’s milk. Each individual case must identify those foods that are responsible.

Seborrhoeic eczema leads to scaling of the scalp and redness of the ears, eyebrows, side of the nose and possibly armpits and groin.

Stasis eczema (or varicose eczema) may arise from varicose vein problems, usually limited to the lower third of the leg.

Discoid eczema has coin-shaped patches preferring extensor surfaces of arms and legs.

Contact eczema may be caused by washing-up detergents, etc. See: CONTACT DERMATITIS.

While emotional or psychic disturbance may worsen, eczema is seldom a psychosomatic disorder arising from stressful situations. Contact with water may worsen. Hairdressers and those allergic to dyes may require patch tests.

Eczema patients, especially atopic, have a metabolic deficiency of linoleic acid (a dietary fatty acid) to y-linolenic acid, which is found in Evening Primrose oil. Eczema may develop in bottle-fed babies due to absence of GLA (gamma-linolenic acid) in commercial powdered milk. GLA is present in Evening Primrose.

A cross-over trial in 99 patients (adults and children) by Bristol (England) dermatologists found Evening Primrose oil (Efamol capsules) produced an overall 43 per cent improvement in eczema severity: doses – 4 to 6 capsules twice daily (adults); 2 capsules twice daily (children). Lower doses were not effective.

Alternatives. Barberry, Bladderwrack, Blood root, Blue Flag root, Bogbean, Burdock, Clivers, Devil’s Claw, Echinacea, Figwort, Fringe Tree, Fumitory, Garlic, Guaiacum, Goldenseal, Mountain Grape, Gotu Kola, Nettles, Plantain, Poke root, Queen’s Delight, Red Clover, Sarsaparilla, Sassafras, Wild Indigo, Heartsease, Yellow Dock.

Tea. Combine herbs: equal parts: Gotu Kola, Clivers, Red Clover. 1-2 teaspoons to each cup boiling water; infuse 5-10 minutes; 1 cup thrice daily, before meals (Dry eczema).

Formula: equal parts, Burdock root, Yellow Dock root, Valerian root. Dose. Liquid Extracts, 1 teaspoon. Tinctures, 1-2 teaspoons. Powders, two 00 capsules or one-third teaspoon. Thrice daily, before meals. Practitioner: specific medication.

Dry eczema. Equal parts, tinctures: Yarrow, Dandelion, Calendula, Echinacea.

Weeping eczema. Combine tinctures: Barberry 1; Clivers 2; Echinacea 2.

Seborrhoeic eczema. Combine tinctures: Blue Flag root 1; Meadowsweet 2; Boneset 1.

Discoid eczema. Combine tinctures: Yellow Dock 2; Mountain Grape 1; Echinacea 1.

Varicose eczema. Combine tinctures: Echinacea 2; Calendula (Marigold) 1; Hawthorn 1.

Dosage for the above: One to two 5ml teaspoons in water thrice daily before meals.

Skin Care. May reduce necessity for steroid creams. It is best to avoid: lanolin and Coconut oil compounds that may contain coal tar. Wash in soft water (rain water) or water not containing chemical softeners.

Indicated: soothing softening herbal lotions, ointments or creams: Marshmallow, Chickweed, Comfrey, Witch Hazel, Aloe Vera gel, Jojoba oil, Evening Primrose oil. For seborrhoeic eczema: Bran Bath or Bran Wash, twice weekly, soapless, followed by Rosemary shampoo. Vitamin E lotion or cream.

Note: A study carried out at the University of Manchester, England, found that children with eczema had significantly low levels of serum zinc than control-cases. (British Journal of Dermatology, 1984, 111, 597)

Evening Primrose oil. For Omega 6 fatty acids.

Diet. Gluten-free. Oily fish: see entry. Avoid cow’s milk, wheat products.

Supplements. Daily. Vitamins: A (7500iu). C (500mg). E (400iu). Bioflavonoids (500mg). Zinc (15mg). Betaine hydrochloride.

Note: The disorder may be due to a deficiency of essential fatty acids (EFAs) brought about by a deficiency of zinc which is necessary for EFA metabolism.

Chinese herbs. A study has shown herbal treatment to be far superior to placebo in clinical trials. British children with (dry) atopic eczema responded favourably to treatment which included the following herbs known as Formula PSE101.

Ledebouriella sesloides, Potentilla chinesis, Anebia clematidis, Rehmannia glutinosa, Peonia lactiflora, Lophatherum gracile, Dictamnus dasycarpus, Tribulus terrestris, Glycyrrhiza uralensis, Schizonepta tenuifolia. Non-toxicity confirms their safety. (Sheeham M et al. “A controlled trial of traditional Chinese medicinal plants in widespread non-exudative atopic dermatitis”, British Journal of Dermatology, 126: 179-184 1992)

When 10 Chinese herbs were analysed by a team at the Great Ormond Street Hospital, London, it was revealed that no single active ingredient or herb was responsible for success. “It was a combination of all 10 herbs that gave the medicine its healing properties.” This is an example of the synergistic effect of combined plant remedies and supports the herbalist’s belief in use of the whole plant. ... eczema




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